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Usajobs.gov resume builders surplus citymd covid
Conflicts of interest comprise financial interests, activities, and relationships within the past 3 years including but not limited to employment, affiliation, grants or funding, consultancies, honoraria or payment, speaker’s bureaus, stock ownership or options, expert testimony, royalties, donation of medical equipment, or patents planned, pending, or issued. If you have no conflicts of interest, check “No potential conflicts of interest” in the box below. The information will be posted with your response.
Not all submitted comments are published. Please see our commenting policy for details. This symptom can be associated with mild COVID, mostly occurs within 5 days after symptom onset, and can persist for a few days to several months after infection resolution. Host genetics, 2 acute inflammation in the olfactory epithelium, 3 local ACE2 expression, 4 and downregulation of olfactory receptors 5 seem to play a role; however, the viral contribution remains to be explored.
Participants were aged 18 years or older and presented with mild symptoms at the time of sample collection for diagnosis. The study was approved by the Brazilian national commission of ethics in research and written informed consent was obtained from all participants.
Clinical and demographic data were obtained through an updated version of the Brazilian National Health System Questionnaire for COVID that was administered by qualified specialists. Viral lineages were attributed to each participant using genomic surveillance data from the state of Rio de Janeiro.
Individuals were recruited when the original lineages B. The study did not include individuals diagnosed during periods when 2 or more lineages co-circulated at a high frequency. Association analyses were conducted using logistic regression models with the original lineage period as the reference group.
Age, sex, viral load, time since symptom onset, hypertension, diabetes, and smoking status were evaluated as potential confounders. A sensitivity analysis including only individuals diagnosed when COVID vaccines were available after February also was performed, adding vaccination status as an additional variable in the logistic regression models.
Because no vaccines were available during the period of the original lineages, the Gamma period was used as the reference group. The analyses were conducted using R version 4. Of the participants with mild COVID in the cohort, reported olfactory dysfunction Table 1 and did not report this symptom.
Olfactory dysfunction was reported by of the participants The prevalence decreased to The odds of olfactory dysfunction were lower for those infected during Gamma adjusted odds ratio [OR], 0. No association was observed during Delta adjusted OR, 0.
The sensitivity analysis found an adjusted OR of 0. This study found that individuals with mild COVID infected during the Gamma and Omicron waves had lower odds of reporting olfactory dysfunction than individuals infected during the period of the original lineages. These results suggest that the type of SARS-CoV-2 variant might be a risk factor for olfactory dysfunction, along with host genetic susceptibility.
Limitations of this study include self-reported outcome, variant attribution according to epidemiological surveillance data, and possible unmeasured confounding.
The present findings highlight the importance of considering SARS-CoV-2 variants when modeling olfactory outcomes and suggest that olfactory dysfunction might not be a hallmark of COVID with certain variants. Published Online: June 24, Conflict of Interest Disclosures: None reported. No compensation was received by any of the contributors to this work. Our website uses cookies to enhance your experience. By continuing to use our site, or clicking “Continue,” you are agreeing to our Cookie Policy Continue.
Download PDF Comment. Table 1. View Large Download. Table 2. Prevalence and 6-month recovery of olfactory dysfunction. PubMed Google Scholar Crossref. COVID—related anosmia is associated with viral persistence and inflammation in human olfactory epithelium and brain infection in hamsters. SARS-CoV-2 entry factors are highly expressed in nasal epithelial cells together with innate immune genes.
Non-cell-autonomous disruption of nuclear architecture as a potential cause of COVID—induced anosmia. Accessed May 13, This JAMA Insights Clinical Update discusses the presumed mechanism of COVID—related olfactory dysfunction and proposes assessment and management strategies, including olfactory training and adjuvant medication treatments. This diagnostic study conducted in a coronavirus disease 19 COVID screening center of a tertiary university hospital assesses the diagnostic value of self-reported and clinically evaluated olfactory dysfunction and gustatory dysfunction in outpatients with mild to moderate or no COVID symptoms.
This prospective study assesses the feasibility of a novel, objective olfactory test as part of an initial screening for COVID in adults with unknown disease status. Yan, MD. This meta-analysis examines the scale of the public health concern of COVID—related chronic olfactory dysfunction.
Amish M. Piccirillo, MD. This cohort study evaluates the association of COVID infection with olfactory bulb by performing an ultrastructural and histopathological analysis of patients with COVID and age- and disease severity—compatible controls. Save Preferences. Privacy Policy Terms of Use. Limit characters. Limit 25 characters. Conflicts of Interest Disclosure Identify all potential conflicts of interest that might be relevant to your comment.
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